Research Article
Cheating the Beta Cells to Delay the Beginning of Type-2 Diabetes Through Artificial Segregation of Insulin
@INPROCEEDINGS{10.1007/978-3-030-24202-2_1, author={Huber Nieto-Chaupis}, title={Cheating the Beta Cells to Delay the Beginning of Type-2 Diabetes Through Artificial Segregation of Insulin}, proceedings={Bio-inspired Information and Communication Technologies. 11th EAI International Conference, BICT 2019, Pittsburgh, PA, USA, March 13--14, 2019, Proceedings}, proceedings_a={BICT}, year={2019}, month={7}, keywords={Diabetes Beta cells Insulin}, doi={10.1007/978-3-030-24202-2_1} }- Huber Nieto-Chaupis
Year: 2019
Cheating the Beta Cells to Delay the Beginning of Type-2 Diabetes Through Artificial Segregation of Insulin
BICT
Springer
DOI: 10.1007/978-3-030-24202-2_1
Abstract
In this paper, we focus in an artificial mechanism to detain the beginning of the type-2 diabetes disease in those identified patients which might to be developing a phase of prediabetes. From purely electrical interactions or Coulomb forces between a deployed nano sensor around of beta cells and Calcium ions, we propose an artificial entrance of Calcium ions inside the beta-cells allowing them to segregate insulin. The electrical interactions between positively charged insulin inside beta cells is the main assumption of this paper. The permanent segregation of insulin fits well inside of the architecture of advanced networks engineering that contemplates the usage of a bio cyber interface. Therefore, the artificial releasing of granules with repulsive electric forces of insulin becomes a manner to cheat beta cells. This might be also seen as an option to avoid the intake of prediabetes y diabetes pharmacology for large periods. Although the view of this work is theoretical and prospective, it is based entirely in closed-form physics equations that sustain the main claim of this paper: electric interactions driven by charged nano particles would be a window to stop the progress of diseases based on the induced or spontaneous deficit of proteins, hormones and cells that are crucially needed to maintain the human homeostasis.
